Drawing upon the theoretical framework of Cooley’s (1902) “looking-glass self” model, the current study examined how affective dimensions of parenting in adolescence contribute to psychopathology in early adulthood through the mediating mechanism of authenticity – one’s level of comfort with being oneself. Using latent profile analysis (LPA), a three-class solution was identified, classifying inadequate, adequate, and optimal profiles of parenting in adolescence. Class membership was used in a multilevel mediation structural equation model to examine longitudinal links with authenticity and psychopathology (e.g., internalizing, externalizing, and substance abuse disorders) in early adulthood. Results demonstrated that optimal compared to inadequate parent-adolescent relationship quality was directly linked to higher levels of authenticity, which in turn, was directly linked to lower levels of all forms of psychopathology in early adulthood. Results also indicated that authenticity fully mediated the link between profiles of parent-adolescent relationship quality (e.g., grade 12) and internalizing, externalizing, and substance abuse disorders in early adulthood (e.g., four years post-college). In conclusion, the current study demonstrated the influence of affective dimensions of parenting profiles in adolescence on the development of psychopathology in early adulthood via the mediating mechanism of authenticity. Moreover, findings from the current study suggest that authenticity is a critical feature shared in common among various forms of psychopathology. Finally, clinical implications are discussed regarding the potential effectiveness of evidence-based psychotherapies aimed at the promotion of authenticity as a mechanism for improving mental health and well-being.

The current study examined the unique influence of emotional childhood abuse on positive and negative aspects of different types of social relationships (e.g., family, spouse/partner, and friends) in midlife and whether genetic variations of the oxytocin receptor gene (OXTR) moderated these associations. Genetic variations in OXTR are measured by single-nucleotide polymorphisms (SNPs), which have been the most substantially studied prospects for explaining individual differences in socio-behavioral phenotypes. Specifically, an SNP, rs53576, involving a guanine (G) to adenine (A) substitution located in the third intron of the OXTR has been associated with fundamental aspects of social processes and behaviors. Compared to A carriers, individuals homozygous for the G allele have enhanced social competencies and tend to elicit more positive responses from social partners, consequently increasing the overall quality of social relationships across the lifespan. However, the G allele of the OXTR has also been associated with greater social sensitivity. In the current study, conducted among a sample of 614 adults in midlife, it was shown that emotional childhood abuse was significantly associated with having less supportive and more strained relationships in midlife. Regarding supportive family relationships, the effect of emotional childhood abuse was moderated by the OXTR rs53576 polymorphism. Specifically, under conditions of more emotional abuse in childhood, individuals homozygous for the G allele had more supportive family relationships in midlife compared to A carriers. Overall, the findings suggest that genetic variations of OXTR rs53576 may be an important candidate in understanding the development of social relationship functioning within the context of negative early life experiences.